Wide QRS tachycardia

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Raja Selvaraj. Professor of Cardiology, JIPMER

06-06-2020

Introduction

Definition

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What are the causes

  • Ventricular tachycardia
  • Supraventricular tachycardia
    • Rate related aberrancy
    • Preexisting bundle branch block
    • Preexcited / antidromic tachycardia

Why important to identify

  • Acute management
  • Long term management

VT vs SVT - Concept

  • Involvement of atria
    • Usually part of tachycardia mechanism (SVT) - exceptions
    • Not part of tachycardia mechanism (VT)
  • Use of specialized conducting system
    • Ventricular activation uses HPS, but HPS conduction abnormalities (SVT with aberrancy)
    • Ventricular activation originates outside of HPS (VT) - exceptions

VT vs Preexcited tachycardia - Concept

  • Both originate in ventricular muscle - Morphology cannot differentiate
  • Differences
    • APs are restricted to specific locations
    • Atrium involved in preexcited tachycardias

General approach

Sequential approach

  • Regularity
  • Atrial activation
  • Morphology
  • Sinus ECG
  • Response to AV blocking drugs / maneuvers

Regularity

  • Sustained monomorphic VT and SVT usually very regular
  • Significant irregularity points to AF

Atrial activation

  • Number of P - less, equal or more
  • VA relationship - 1:1, other VA conduction, dissociation
  • Morphology of P - sinus / retrograde

Morphology

  • QRS width
  • LBBB like / RBBB like
  • Frontal axis
  • Morphology in V1 and V6
  • Early activation vs late activation

Morphology - Terminology

QRS_nomenclature.png

Morphology - Identify typical patterns

  • RBBB/LBBB, inferior axis, qS aVL and aVR - Outflow tract VT
  • RBBB / LAD, relatively narrow QRS - ILVT
  • LBBB / LAD - ARVC / Atriofascicular (Mahaim) pathway

Sinus ECG

  • Evidence of MI - Correlate with VT origin
  • ARVC
  • Bundle branch block - SVT with aberrancy / BBRT
  • Preexcitation - AVRT with aberrancy / preexcited tachycardia

Adenosine

  • Termination
  • Transient slowing with AV block
  • No change

Examples

52 M with palpitations

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Dissociated, slower sinus rhythm

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35 F, repaired TOF

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After treatment with Dilzem

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35 M, Normal heart

thangavel ecg sca004.jpg

thangavel ecg sca004_m.jpg

33 M with palpitations

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35 M with palpitations

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ECG in sinus

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60 M

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Adenosine

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Dilzem

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34 F, Palpitations and syncope

ecg_preecited_af-0.jpg

Sinus rhythm

ecg_preecited_af-1.jpg

54 M, known CAD - old AWMI

kumar old mi sustained VT_001.jpg

60 M, old AWMI

RAMADASS awmi  svt CHANGES001.jpg

Adenosine

RAMADASS awmi  svt CHANGES002.jpg

Elderly male

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30 F with paroxysmal palpitations

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Sinus ECG

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Summary

  • WQRST - Can be VT or SVT
  • Atrial activation is key - A < V in VT, A > V is SVT
  • Morphology when A = V, but not very reliable
  • Learn in terms of concepts more than rules
  • Adenosine in stable tachycardia